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- https://bio.libretexts.org/Courses/Folsom_Lake_College/BIOL_440%3A_General_Microbiology_(Panoutsopoulos)/06%3A_The_Immune_System_Nonspecific_and_Specific_Defenses/6.02%3A_Innate_Nonspecific_Host_Defenses/6.2.05%3A_Inflammation_and_FeverInflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infect...Inflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infection. Chronic inflammation occurs when the inflammatory response is unsuccessful, and may result in the formation of granulomas (e.g., with tuberculosis) and scarring (e.g., with hepatitis C viral infections and liver cirrhosis).
- https://bio.libretexts.org/Courses/Manchester_Community_College_(MCC)/Remix_of_Openstax%3AMicrobiology_by_Parker_Schneegurt_et_al/13%3A_Innate_Nonspecific_Host_Defenses/13.4%3A_Inflammation_and_Fevernflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infecti...nflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infection. Chronic inflammation occurs when the inflammatory response is unsuccessful, and may result in the formation of granulomas (e.g., with tuberculosis) and scarring (e.g., with hepatitis C viral infections and liver cirrhosis).
- https://bio.libretexts.org/Courses/City_College_of_San_Francisco/Introduction_to_Microbiology_(Liu_et_al.)/21%3A_Innate_Immunity_II/21.05%3A_InflammationInflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infect...Inflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infection. Chronic inflammation occurs when the inflammatory response is unsuccessful, and may result in the formation of granulomas (e.g., with tuberculosis) and scarring (e.g., with hepatitis C viral infections and liver cirrhosis).
- https://bio.libretexts.org/Courses/Mansfield_University_of_Pennsylvania/BSC_3271%3A_Microbiology_for_Health_Sciences_Sp21_(Kagle)/14%3A_Immunology/14.01%3A_Innate_Nonspecific_Host_Defenses/14.1.05%3A_Inflammation_and_Fevernflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infecti...nflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infection. Chronic inflammation occurs when the inflammatory response is unsuccessful, and may result in the formation of granulomas (e.g., with tuberculosis) and scarring (e.g., with hepatitis C viral infections and liver cirrhosis).
- https://bio.libretexts.org/Courses/Sacramento_City_College/BIOL_440%3A_General_Microbiology_(Hughes)/13%3A_Week_13/21%3A_Innate_Nonspecific_Host_Defenses_(Part_B)/21.03%3A_Inflammation_and_Fevernflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infecti...nflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infection. Chronic inflammation occurs when the inflammatory response is unsuccessful, and may result in the formation of granulomas (e.g., with tuberculosis) and scarring (e.g., with hepatitis C viral infections and liver cirrhosis).
- https://bio.libretexts.org/Courses/Clinton_College/BIO_403%3A_Microbiology_(Neely)/06%3A_The_Immune_System_Nonspecific_and_Specific_Defenses/6.02%3A_Innate_Nonspecific_Host_Defenses/6.2.05%3A_Inflammation_and_FeverInflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infect...Inflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infection. Chronic inflammation occurs when the inflammatory response is unsuccessful, and may result in the formation of granulomas (e.g., with tuberculosis) and scarring (e.g., with hepatitis C viral infections and liver cirrhosis).
- https://bio.libretexts.org/Courses/North_Central_State_College/BIOL_1550%3A_Microbiology_(2025)/21%3A_Innate_Immunity_II/21.06%3A_InflammationInflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infect...Inflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infection. Chronic inflammation occurs when the inflammatory response is unsuccessful, and may result in the formation of granulomas (e.g., with tuberculosis) and scarring (e.g., with hepatitis C viral infections and liver cirrhosis).
- https://bio.libretexts.org/Courses/New_England_College/Microbiology_with_NEC/12%3A_Innate_Nonspecific_Host_Defenses/12.05%3A_Inflammation_and_Fevernflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infecti...nflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infection. Chronic inflammation occurs when the inflammatory response is unsuccessful, and may result in the formation of granulomas (e.g., with tuberculosis) and scarring (e.g., with hepatitis C viral infections and liver cirrhosis).
- https://bio.libretexts.org/Courses/North_Central_State_College/BIOL_1550%3A_Microbiology_(2025)/21%3A_Innate_Immunity_II/21.07%3A_FeverInflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infect...Inflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infection. Chronic inflammation occurs when the inflammatory response is unsuccessful, and may result in the formation of granulomas (e.g., with tuberculosis) and scarring (e.g., with hepatitis C viral infections and liver cirrhosis).
- https://bio.libretexts.org/Courses/Prince_Georges_Community_College/PGCC_Microbiology/06%3A_Immunology/6.02%3A_Non-Adaptive_Immunity/6.2.06%3A_Inflammation_and_FeverInflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infect...Inflammation results from the collective response of chemical mediators and cellular defenses to an injury or infection. Acute inflammation is short lived and localized to the site of injury or infection. Chronic inflammation occurs when the inflammatory response is unsuccessful, and may result in the formation of granulomas (e.g., with tuberculosis) and scarring (e.g., with hepatitis C viral infections and liver cirrhosis).
- https://bio.libretexts.org/Courses/City_College_of_San_Francisco/Introduction_to_Microbiology_(Liu_et_al.)/21%3A_Innate_Immunity_II/21.06%3A_FeverThis page explains fever as an inflammatory response that raises body temperature to help fight infections, triggered by pyrogens. The hypothalamus regulates this process through mechanisms like vasoc...This page explains fever as an inflammatory response that raises body temperature to help fight infections, triggered by pyrogens. The hypothalamus regulates this process through mechanisms like vasoconstriction and shivering, while cooling occurs during a crisis phase through vasodilation and sweating. Although fever can enhance immune response, excessive heat may lead to tissue damage or become life-threatening, especially with bacterial superantigens.
